How Zyatonix Works

We have takena three-pronged approach in engineering Zyatonix. All three angles represent a quantumleap in weight-loss technology, however it Zyatonix’s ability to actually trigger andcause fat cell death – literally kills fat cells in your body and thereby removingthem forever – something that was only previously achieved through liposuction –that really sets Zyatonix apart from any and all weight-loss pills ever developed. Here arethe three-pronged attack of Zyatonix:

• Fat CellDeath –When you get rid of fat cells – they don’t come back.

• LipidMobilization– Technology that allows you to burn body fat without exercise.

• SerotoninEnhancement & Appetite Suppression – Delivery system that shutsdown your appetite while elevating your sense of satisfaction and fullfillment.


Here is a more detailed explanation of how Zyatonix works and why it produces suchincredible.

IncinerateFat – SayGoodbye to your Love handles!

Fat mobilization is a new breakthrough in the field of weight loss. The newest thermogenicbreakthrough introduced into the market is called Lipid Mobilization, which helps you burnbody fat without exercise. In vitro research shows that lipid mobilization is one of theprocesses that release fat into the bloodstream to be burned as energy. On the fat cell’ssurface are receptors that signal the cell to hold stored fat. Natural Alpha-2 antagonistshave been shown to switch off these receptors. Freed fatty acids can then move out of thecell and into the bloodstream. These released fats are shuttled away from fat andprevented from simply being re-deposited. This is especially effective in the stubbornabdominal and hip areas of both men and women. Best of all, when taken before exercise,fat mobilizers are shown to boost lipolysis ( the process of mobilizing fats from cells)and increase blood serum free fatty acid levels both during and after exercise.

The extent of which exercise burns body fat is totally dependent upon the rate oflipolysis. Lipolysis is the rate at which fat is mobilized from fat cells and enters thebloodstream as free fatty acids (FFAs). Exercise triggers lipolysis, and highly trainedindividuals possess a greater lipolytic rate, an ability to “burn” more fatduring exercise, than untrained people. If you want the greatest possible fat burningeffect from exercise, then increasing your rate of lipolysis is the way to do it. A highrate of lipolysis ensures greater fat mobilization by the liver and mitochondria in musclecells during exercise. In turn, this process ensures that a greater concentration of bodyfat is burned during exercise. The impact of fat mobilizers on post-exercise fatmetabolism is particularly evident 30 minutes after cardio exercise. One study showed thatFFA levels in the bloodstream doubled in those that took a fat mobilizer prior to exercisecompared to a placebo.

Many physiological factors stimulate and inhibit the breakdown of adipose tissue into freefatty acids and glycerol and their mobilization into the bloodstream to be used as fuel byother cells and tissues. Fasting, feeding, exercise, and stress have pronounced and rapideffects on lipolysis via hormones and other endogenous substances. As well, clinicalconditions such as diabetes and obesity are associated with alterations in lipolysis. Ageand gender are also of importance. Insulin and catecholamines are the main regulatoryhormones of lipid mobilization. Insulin is the major antilipolytic hormone because of itseffects on enzymes within the adipocyte. Insulin also enables the entry of glucose intothe cells by inducing glucose transporter activity. Glucose serves as the backbone for theglycerol molecule to which fatty acids attach and form triacylglycerols. Thecatecholamines serve a dual function. You must first become acquainted with fat cellbiology to comprehend the regulation of fat loss. Lipogenesis and lipolysis can beconsidered the Yin and Yang of adipose tissue metabolism. Lipogenesis is the process offat accumulation and lipolysis is that of fat breakdown and release into the bloodstream.

FatMobilizers and Their Unique Fat Burning Abilities

There arephysiological differences in fat cells, depending upon where they are located in the body.Fat cells located in the gut (visceral adipocytes) differ from fat cells located in thelower regions of the body (hips, thighs, lovehandles). Fat cells within the stomachcontain a lot of beta-receptors. These cells respond to release fat when stimulated by theclassic “fat burners,” such as caffeine, ephedrine, and synephrine. Thesecompounds stimulate lipolysis specifically by increasing norepinephrine delivery to thevisceral fat cells and catecholamine secretion that activates the beta-receptors andincreases cAMP within cells. However, fat cells located around the hips, and lovehandlescharacteristically contain very few beta-receptors and respond poorly to catecholaminerelease that is induced by exercise and beta-stimulants. However, these lower body fatcells contain a lot of alpha adrenoreceptors. Alpha-receptors are tricky and obstinate ifyou want them to release their fat stores. When stimulated, these receptors activate otherproteins that inhibit adenylcyclase, thus antagonizing the ability of beta-adrenoreceptorsto boost cAMP generation, and therefore, shut down the usual fat mobilization process.Basically, when taking caffeine, ephedrine, and synephrine supplements in an effort tostimulate fat loss, the alpha-receptors on lower body fat cells say, “No! No fatmobilization for you!” Fat cells within the lower half of the body contain a higherconcentration of alpa-receptors and lower concentration of beta-receptors. Therefore, theyare quite resistant to lipolysis. Women characteristically carry more fat on the hips andthighs than men do, and this difference in fat cell structure is one reason why most womenhave a tougher battle with fat loss … Until Now!