How Zyatrim Works

We have taken a three-pronged approach in engineering Zyratrim. All three angles represent a quantum leap in weight-loss technology, however it Zyatrim’s ability to actually trigger and cause fat cell death – literally kills fat cells in your body and thereby removing them forever – something that was only previously achieved through liposuction – that really sets Zyatrim apart from any and all weight-loss pills ever developed. Here are the three-pronged attack of Zyatrim:

• Fat Cell Death – When you get rid of fat cells – they don’t come back.

• Lipid Mobilization – Technology that allows you to burn body fat without exercise.

• Serotonin Enhancement & Appetite Suppression – Delivery system that shuts down your appetite while elevating your sense of satisfaction and fullfillment.


Here is a more detailed explanation of how Zyatrim works and why it produces such incredible.

Incinerate Fat – Say Goodbye to your Love handles!

Fat mobilization is a new breakthrough in the field of weight loss. The newest thermogenic breakthrough introduced into the market is called Lipid Mobilization, which helps you burn body fat without exercise. In vitro research shows that lipid mobilization is one of the processes that release fat into the bloodstream to be burned as energy. On the fat cell’s surface are receptors that signal the cell to hold stored fat. Natural Alpha-2 antagonists have been shown to switch off these receptors. Freed fatty acids can then move out of the cell and into the bloodstream. These released fats are shuttled away from fat and prevented from simply being re-deposited. This is especially effective in the stubborn abdominal and hip areas of both men and women. Best of all, when taken before exercise, fat mobilizers are shown to boost lipolysis ( the process of mobilizing fats from cells) and increase blood serum free fatty acid levels both during and after exercise.

The extent of which exercise burns body fat is totally dependent upon the rate of lipolysis. Lipolysis is the rate at which fat is mobilized from fat cells and enters the bloodstream as free fatty acids (FFAs). Exercise triggers lipolysis, and highly trained individuals possess a greater lipolytic rate, an ability to “burn” more fat during exercise, than untrained people. If you want the greatest possible fat burning effect from exercise, then increasing your rate of lipolysis is the way to do it. A high rate of lipolysis ensures greater fat mobilization by the liver and mitochondria in muscle cells during exercise. In turn, this process ensures that a greater concentration of body fat is burned during exercise. The impact of fat mobilizers on post-exercise fat metabolism is particularly evident 30 minutes after cardio exercise. One study showed that FFA levels in the bloodstream doubled in those that took a fat mobilizer prior to exercise compared to a placebo.

Many physiological factors stimulate and inhibit the breakdown of adipose tissue into free fatty acids and glycerol and their mobilization into the bloodstream to be used as fuel by other cells and tissues. Fasting, feeding, exercise, and stress have pronounced and rapid effects on lipolysis via hormones and other endogenous substances. As well, clinical conditions such as diabetes and obesity are associated with alterations in lipolysis. Age and gender are also of importance. Insulin and catecholamines are the main regulatory hormones of lipid mobilization. Insulin is the major antilipolytic hormone because of its effects on enzymes within the adipocyte. Insulin also enables the entry of glucose into the cells by inducing glucose transporter activity. Glucose serves as the backbone for the glycerol molecule to which fatty acids attach and form triacylglycerols. The catecholamines serve a dual function. You must first become acquainted with fat cell biology to comprehend the regulation of fat loss. Lipogenesis and lipolysis can be considered the Yin and Yang of adipose tissue metabolism. Lipogenesis is the process of fat accumulation and lipolysis is that of fat breakdown and release into the bloodstream.

Fat Mobilizers and Their Unique Fat Burning Abilities

There are physiological differences in fat cells, depending upon where they are located in the body. Fat cells located in the gut (visceral adipocytes) differ from fat cells located in the lower regions of the body (hips, thighs, lovehandles). Fat cells within the stomach contain a lot of beta-receptors. These cells respond to release fat when stimulated by the classic “fat burners,” such as caffeine, ephedrine, and synephrine. These compounds stimulate lipolysis specifically by increasing norepinephrine delivery to the visceral fat cells and catecholamine secretion that activates the beta-receptors and increases cAMP within cells. However, fat cells located around the hips, and lovehandles characteristically contain very few beta-receptors and respond poorly to catecholamine release that is induced by exercise and beta-stimulants. However, these lower body fat cells contain a lot of alpha adrenoreceptors. Alpha-receptors are tricky and obstinate if you want them to release their fat stores. When stimulated, these receptors activate other proteins that inhibit adenylcyclase, thus antagonizing the ability of beta-adrenoreceptors to boost cAMP generation, and therefore, shut down the usual fat mobilization process. Basically, when taking caffeine, ephedrine, and synephrine supplements in an effort to stimulate fat loss, the alpha-receptors on lower body fat cells say, “No! No fat mobilization for you!” Fat cells within the lower half of the body contain a higher concentration of alpa-receptors and lower concentration of beta-receptors. Therefore, they are quite resistant to lipolysis. Women characteristically carry more fat on the hips and thighs than men do, and this difference in fat cell structure is one reason why most women have a tougher battle with fat loss … Until Now!